13 PDF Article

RESUMEN La corea es un trastorno del movimiento que tiene a la eritrocitosis como causa poco frecuente. La eritrocitosis o poliglobulia es el aumento de la. fisiopatologúia del sistema hematopoyético hematopoyesis proceso de creación de células sanguíneas maduras mielopoyesis: creación de eritropoyesis. Infografía más información ampliada del recuento de leucocitos, alteraciones por exceso o por defecto y causas más habituales.

Author: Shajas Maujin
Country: Uruguay
Language: English (Spanish)
Genre: Finance
Published (Last): 3 September 2014
Pages: 50
PDF File Size: 11.76 Mb
ePub File Size: 8.64 Mb
ISBN: 274-9-29463-384-5
Downloads: 86986
Price: Free* [*Free Regsitration Required]
Uploader: Kajind

Therefore, the exact cause of erytrocytosis remains unclear. Access nearly titles, over 4 million cited references, and open access with links to full text through a local language interface with an easy search experience. Follow-up was completed for Patients. Prognostic value of the hematocrit in patients with severe COPD receiving long-term oxygen therapy.

Leucocitos o Serie Blanca. Alteraciones y Causas.

Oxyhemoglobin dissociation curve P50 was normal and Janus kinase 2 JAK-2 mutation analysis was negative, ruling out polycythemia vera. G47R barttin and CLC-Ka may result in a less severe reduction of chloride currents, as seen in missense mutations, enabling eritrocitossi to retain some residual function with CLC-Kb, conditioning a milder phenotype.

Conforme descrito por Brum et al. Bartter syndrome, Hypokalemia, Chloride channels, Case reports.

Exacerbation rate, health status and mortality in COPD – a review of potential interventions. Even though exacerbations are the main cause of eeitrocitosis consultation in patients suffering from lung diseases, erythrocyte parameters are not assessed in their prognosis.

Variables were described using an additional categorization by hematocrit levels and an adjustment in a multivariate model through logistic regression. Clinical audit indicators of outcome following admission to hospital with acute exacerbation of chronic obstructive pulmonary disease.

Directory of Open Access Journals DOAJ increases the visibility and ease of use of open access scientific and scholarly journals, aims to be ccausas and cover all that journals that use a quality control system to guarantee the content.

  EN 10277-2 PDF

Revista de la Facultad de Medicina

Nihon Naibunpi Gakkai Zasshi. To establish a possible relationship between the different hematocrit levels with a day prognosis in patients admitted with exacerbated chronic lung disease and hypoxemia. Hemorheology in the erythrocytoses.

The frequency of anemia was 7. Support Center Support Center. Repetitive hemodilution in chronic obstructive pulmonary disease and pulmonary hypertension: Mortality and mortality-related factors after hospitalization for acute exacerbation of COPD. It is currently being managed in Colombia by the Universidad Nacional de Colombia.

It’s a one stop shop for users of OA Journals. The most intriguing feature of the present case was the presence of a marked erythrocitosis in a non-smoking patient, in the absence causad polycythemia vera, JAK-2 mutations or other causes of primary polycythemia. Eritocitosis and view critically important regional content with international impact to get a comprehensive picture of the influencers and drivers of regional research.

This research could not determine any relationship between erythrocyte parameters and prognosis of patients suffering from pulmonary diseases; nevertheless, extreme values of hematocrits tended to have adverse outcomes. The patient had other deaf brother and a stone-forming sister. Antenatal Bartter syndrome BS type IV is characterized by polyhydramnios, premature birth, sensorineural deafness, severe salt and water loss, in the perinatal period, hypokalemic alkalosis, fever, vomiting, diarrhea, failure to thrive and chronic renal failure developing during infancy.

Our patient did not show this phenotype, which agrees with other reports 23 possibly because of the low serum ionized calcium dee led to a lower filtered load of calcium. La frecuencia de anemia fue de 7.


Get a more complete global picture by discovering new insights from research in Latin America, Spain, Portugal, the Caribbean and South Africa.

These findings are in agreement with Vaisbich et al. Latindex Latindex is the product of cooperation by a network of Latin-American institutions operating in a coordinated way to gather and disseminate bibliographic information about serialised scientific publications produced in the region.


Assim, a causa exata da eritrocitose permanece desconhecida. Symptomatic and puomonary response to acute phlebotomy in secondary polycythemia. DOAJ increases the visibility and ease of use of open access scientific and scholarly journals, aims to be comprehensive and cover all that journals that use a quality control system to guarantee the content. However, the major source of EPO synthesis in the kidney is presently known to be the interstitial fibroblasts and not the juxtaglomerular apparatus.

In addition to phosphaturia, another evidence of proximal tubular dysfunction in the current case was the increased level of urinary RBP, a low molecular weight protein. The etiology of erythrocytosis was investigated. Besides, EPO levels showed to be within normal range in the present case. Excluiu-se nefrocalcinose, por meio de tomografia computadorizada helicoidal.

Serum 25OH – vitamin D Serum iron, ferritin and transferrin were normal. Find articles by Ita Pfeferman Heilberg. A study based on a day follow-up was conducted. Finally, after a 2-month course of oral cholecalciferol supplementation 50,UIPTH levels normalized, suggesting that high PTH might have been secondary to the mild hypocalcemia and sub-normal levels of 25OH – vitamin D.

In the present case, renal function was preserved, like in all other described patients carrying this mutation. Although the finding of mild hypophosphatemia and elevated serum intact PTH could have initially suggested the presence of some disorder of phosphate metabolism in the present case, the association of hypokalemia and hearing impairment, even in the absence of metabolic alkalosis, led us to hypothesize about a late onset presentation of BS type IV.